Dmitry, I need your help, I’m trying understand something and to explain it simply and coherently. You shared an article a while back on Telegram: ‘CD38 Inhibits Prostate Cancer Metabolism and Proliferation by Reducing Cellular NAD+Pools’. At first glance to a relatively uninformed person, this study might appear to be saying that because cancer cells utilize NAD+ to proliferate, then that should raise alarm bells about the whole idea of increasing NAD+ as a therapy for cancer. But, I know that is wrong, having read and tried to understand the extensive research that you’ve shared, and my imperfect understanding is that cancer cells can only utilize NAD+ when it is produced by the salvage pathway of NAD+ production, whereas NAD+ produced by the the nicotinic acid pathway (Preiss-Handler pathway) is only utilized by healthy and non-cancer cells and not used by cancer cells. I’m asking you if my understanding is correct and also if what I described is always the case for all cancers and I’m also hoping you can explain a little bit of the why and how of why and how that works, the research studies leave me feeling lost in the weeds and unable to form a simple streamlined explanation. Thank you very much